Felipe Cabral Miranda

Instituição:

Universidade Federal do Rio de Janeiro

Centro:

Centro de Ciências da Saúde

Unidade:

Instituto de Ciências Biomédicas

Departamento:

Técnico Administrativos/ICB

ORCID:

não disponível no Lattes


Formação:
  • Universidad de Chile

    | Pós-Doutorado | 2018 - 2020
  • Universidade de Chile

    | Pós-Doutorado | 2016 - 2018
  • Universidade Federal do Rio de Janeiro

    Ciencias Biológicas | Doutorado | 2011 - 2015
  • Universidade Federal Fluminense

    Neuroimunologia | Mestrado | 2010 - 2011
  • Universidade Federal Fluminense

    Biomedicina | Graduação | 2006 - 2009
Laboratórios:
Nuvens de Palavras:
Artigos:

(100.00% artigos com DOI)

Titulo DOI Ano
Age-Associated Upregulation of Glutamate Transporters and Glutamine Synthetase in Senescent Astrocytes In Vitro and in the Mouse and Human Hippocampus 10.1177/17590914231157974 2023
Preventing brain aging by the artificial enforcement of the unfolded protein response 10.4103/1673-5374.377608 2023
The unfolded protein response transcription factor XBP1s ameliorates Alzheimer?s disease by improving synaptic function and proteostasis 10.1016/j.ymthe.2023.03.028 2023
Unfolded protein response / signaling is required for healthy mammalian brain aging 10.15252/embj.2022111952 2022
Proteostasis deregulation as a driver of C9ORF72 pathogenesis 10.1111/jnc.15529 2021
Mutation in protein disulfide isomerase A3 causes neurodevelopmental defects by disturbing endoplasmic reticulum proteostasis 10.15252/embj.2020105531 2021
Progenitor death drives retinal dysplasia and neuronal degeneration in a mouse model of Atrip-Seckel syndrome 10.1242/dmm.045807 2020
ER stress links aging to sporadic ALS 10.18632/aging.101705 2019
Interplay Between the Unfolded Protein Response and Immune Function in the Development of Neurodegenerative Diseases 10.3389/fimmu.2018.02541 2018
Endoplasmic reticulum proteostasis impairment in aging 10.1111/acel.12599 2017
ER stress in neurodegenerative disease: from disease mechanisms to therapeutic interventions 10.1515/ersc-2017-0002 2017
Commentary: XBP-1 Is a Cell-Nonautonomous Regulator of Stress Resistance and Longevity 10.3389/fnagi.2016.00182 2016
rAAV8-733-Mediated Gene Transfer of CHIP/Stub-1 Prevents Hippocampal Neuronal Death in Experimental Brain Ischemia 10.1016/j.ymthe.2016.11.017 2016
CHIP, a carboxy terminus HSP-70 interacting protein, prevents cell death induced by endoplasmic reticulum stress in the central nervous system 10.3389/fncel.2014.00438 2015
Are the mechanisms driving somatosensory reorganization cortical or subcortical? 10.3389/fnana.2014.00062 2014
A time-dependent effect of caffeine upon lesion-induced plasticity 10.1016/j.neures.2011.05.018 2011
Eventos:

(0.00% eventos com DOI)

Titulo DOI Ano
Tratamento com cafeína altera a expressão de receptores purinérgicos no colículo superior e o desenvolvimento da via retinotectal 2010
Caffeine and neuronal plasticity: the influence of purinergic modulation in CNS 2010
Alteration in A1 receptor expression in the superior colliculus of rats: potencial correlation between purinergic receptors 2010
CAFFEINE MODULATE RETINAL-LESION INDUCED PLASTICITY IN THE RETINOTECTAL PATHWAY 2009
A FUNCTIONAL ROLE OF A1 AND A2A ADENOSINE RECEPTORS IN THE DEVELOPMENT OF RETINOCOLLICULAR TOPOGRAPHY 2009
TRATAMENTO SUB-CRÕNICO COM CAFEÍNA PROMOVE EXPANSÃO DOS AXÔNIOS RETINOTECTAIS E 2009
A LOCAL BLOCKADE OF A1 ADENOSINE RECEPTOR DISRUPTS RETINOTECTAL 2008
THE ROLE FOR ADENOSINE A2A RECEPTOR IN THE STABILIZATION OF RETINOTECTAL TOPOGRAPHY 2008
Neuroplasticity in the visual system: A possible role of purinergic receptors and glial cells activation 2008
Publicações:
Minha Rede: