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Palavras-chave
cancer macrophage proximal tubule apoptotic sialidase injury structure maternal histone deacetylase glycan corynebacterium diphtheriae hyperglycemia induces atividade atpasica biosynthetic glioblastoma atpase activity protein kinase dopamina trans sialidase hidroxilase cell kidney tubule cell plasmodium falciparum immunity celulas glcnacylation receptor pathway